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Individuals affected by GM-1 gangliosidoses have very similar symptoms to those with Tay-Sachs or Sandhoff. GM-1 is caused by a deficiency of beta-galactosidase, which leads to an accumulation of gangliosides within specific tissues of the body. This disorder causes progressive neurological damage and is inherited in an autosomal recessive fashion. GM-1 has three forms: Classic Infantile, Juvenile and Late Onset or Chronic GM-1.
It is important to note that GM-1 ‘breeds true in a family,’ which means if a family has a child affected by Classic Infantile GM-1, then their other children will only be at risk for having Classic Infantile GM-1. One set of parents could not have a child affected by Classic Infantile and Juvenile or Late Onset GM-1.
Causes
The GM-1 gangliosidoses are caused by a deficiency of the beta-galactosidase (GLB1) enzyme; the gene for this enzyme is located on chromosome 3. This enzyme breaks down carbohydrates into parts so they can be recycled.
Testing
Carrier testing and prenatal testing for GM-1 are available. Visit the Testing section of the site to learn more about carrier testing. For a list of laboratories capable of GM-1 carrier testing please visit www.genetests.org.
The following online resources may be helpful in learning more about GM-1 disease:
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